Tag: digestion and absorption

The liver stores glycogen but does NOT release glycogen into blood. Instead, it performs glycogenolysis to break down glycogen and releases glucose into blood. All other options ARE liver functions: bile production, bilirubin uptake/excretion, and urea synthesis.

Bile juice contains bile salts and pigments but lacks digestive enzymes. It aids digestion through the physical emulsification of fats.

The falciform ligament is a fold of peritoneum that attaches the liver to the anterior abdominal wall and the diaphragm.

An antioxidant is a molecule that inhibits the oxidation of other molecules, thereby protecting cells from damage caused by free radicals.

Bile salts (like sodium glycocholate) lower the surface tension of fat droplets, breaking them into smaller particles to increase the surface area for lipase action.

Nicotinamide is indeed a vitamin (B3). Crypts of Lieberkuhn are in the small intestine, not the liver. Steapsin is pancreatic lipase, not amylase. Thus, statements II and III are incorrect.

The liver is responsible for the urea cycle, which converts toxic ammonia into urea. If the liver is removed, ammonia cannot be processed and will accumulate in the blood.

The liver acts as a modulator by storing or releasing glucose (glycogenolysis/glycogenesis), while the islets of Langerhans (pancreas) act as effectors by secreting insulin and glucagon to control blood sugar levels.

The liver is the primary site of the urea cycle, where ammonia (a toxic byproduct of protein metabolism) is converted into urea for excretion.