Cancer Genetics

Cancer cells exhibit uncontrolled cell division, leading to the formation of tumors.

Point mutations can activate oncogenes by introducing changes in the protein structure or function.

APC (Adenomatous Polyposis Coli) is a tumor suppressor gene frequently mutated in colorectal cancer.

DNA repair mechanisms play a crucial role in preventing cancer by correcting errors that may lead to mutations.

Li-Fraumeni syndrome is an inherited cancer predisposition syndrome associated with an increased risk of various types of cancer.

Tumor suppressor genes play a critical role in preventing cancer by inducing apoptosis (programmed cell death) in damaged or abnormal cells.

Translocation, the exchange of genetic material between two chromosomes, is a common chromosomal rearrangement in cancer cells.

Carcinogenesis refers to the process by which normal cells undergo a series of genetic and molecular changes, leading to the development of cancer.

Leukemia is a type of cancer that arises from the uncontrolled proliferation of blood cells in the bone marrow and lymphatic system.

The BRCA1 gene encodes a protein involved in DNA repair and maintaining genomic stability.

Sarcomas are cancers that originate from the connective tissues of the body.

Metastasis is the process by which cancer cells spread from the primary tumor to other parts of the body through the bloodstream or lymphatic system.

Hormone therapy is a targeted therapy that involves the use of hormones to block the growth of hormone-sensitive cancers.

The TP53 gene encodes the p53 protein, which plays a crucial role in preventing cancer by regulating cell cycle progression and inducing apoptosis.

Carcinomas are the most common type of cancer and arise from epithelial tissues.